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Evaluating the relationship of CD101 and UBE2V1 to genital mucosal inflammation

While it is well known that genital tract inflammation increases the risk of sexually acquiring HIV-1, the mechanism for this is poorly understood. We recently found that variants in CD101 and UBE2V1, genes that may modulates different aspects of inflammation, also contribute to increased risk of HIV-1 infection. We propose to collect genital mucosal samples to determine if these gene variants are associated with changes in genital mucosal inflammation either at the initial visit, in the context of sexually transmitted infections, or with changes in sexual exposure.

Active Dates 
07/14/2017 to 06/30/2022
Faculty Involved